Neuroscientist Says Protein Could Prevent Secondary Damage After Stroke
One of two proteins that regulate nerve cells and assist in overall brain function may be the key to preventing long-term damage as a result of a stroke, the leading cause of disability and third leading cause of death in the United States.
In a recent study published in theJournal of Neuroscience, Bonnie Firestein, professor of cell biology and neuroscience, in the School of Arts and Sciences, says the new research indicates that increased production of two proteins — cypin and PSD-95 — results in very different outcomes.
While cypin — a protein that regulates nerve cell and neuron branching critical to normal brain functioning — prevents nerve cells not damaged during the initial stroke from losing the ability to communicate with other cells and halts any secondary brain or neurological damage, PSD-95 accelerates cell destruction and inhibits recovery. Secondary injury from a stroke can occur days or even weeks after the injury and often includes a lack of blood flow, insufficient oxygen, and swelling of the brain.
Read more (Via Science Daily)
Stroke image from: defencepa.com